Authors

Bina Joe, College of Medicine and Life Sciences
Yasser Saad, College of Medicine and Life Sciences
Norman H. Lee, The George Washington University School of Medicine and Health Sciences
Bryan C. Frank, The George Washington University School of Medicine and Health Sciences
Ovokeraye H. Achinike, The George Washington University School of Medicine and Health Sciences
Truong V. Luu, The George Washington University School of Medicine and Health Sciences
Kathirvel Gopalakrishnan, College of Medicine and Life Sciences
Edward J. Toland, College of Medicine and Life Sciences
Phyllis Farms, College of Medicine and Life Sciences
Shane Yerga-Woolwine, College of Medicine and Life Sciences
Ezhilarasi Manickavasagam, College of Medicine and Life Sciences
John P. Rapp, College of Medicine and Life Sciences
Michael R. Garrett, College of Medicine and Life Sciences
David Coe, Cleveland Clinic Foundation
Suneel S. Apte, Cleveland Clinic Foundation
Tuomo Rankinen, Pennington Biomedical Research Center
Louis Pérusse, Université Laval
Georg B. Ehret, Johns Hopkins University School of Medicine
Santhi K. Ganesh, National Human Genome Research Institute (NHGRI)
Richard S. Cooper, Stritch School of Medicine
Ashley O'Connor, Johns Hopkins University School of Medicine
Treva Rice, Washington University School of Medicine in St. Louis
Alan B. Weder, University of Michigan Medical School
Aravinda Chakravarti, Johns Hopkins University School of Medicine
Dabeeru C. Rao, Washington University School of Medicine in St. Louis
Claude Bouchard, Pennington Biomedical Research Center

Document Type

Article

Publication Date

7-24-2009

Abstract

A previously reported blood pressure (BP) quantitative trait locus on rat Chromosome 1 was isolated in a short congenic segment spanning 804.6 kb. The 804.6 kb region contained only two genes, LOC306664 and LOC306665. LOC306664 is predicted to translate into A Disintegrin-like and Metalloproteinase with Thrombospondin Motifs-16 (Adamts16). LOC306665 is a novel gene. All predicted exons of both LOC306664 and LOC306665 were sequenced. Non-synonymous variants were identified in only one of these genes, LOC306664. These variants were naturally existing polymorphisms among inbred, outbred and wild rats. The full-length rat transcript of Adamts16 was detected in multiple tissues. Similar to ADAMTS16 in humans, expression of Adamts16 was prominent in the kidney. Renal transcriptome analysis suggested that a network of genes related to BP was differential between congenic and S rats. These genes were also differentially expressed between kidney cell lines with or without knock-down of Adamts16. Adamts16 is conserved between rats and humans. It is a candidate gene within the homologous region on human Chromosome 5, which is linked to systolic and diastolic BP in the Quebec Family Study. Multiple variants, including an Ala to Pro variant in codon 90 (rs2086310) of human ADAMTS16, were associated with human resting systolic BP (SBP). Replication study in GenNet confirmed the association of two variants of ADAMTS16 with SBP, including rs2086310. Overall, our report represents a high resolution positional cloning and translational study for Adamts16 as a candidate gene controlling BP. © 2009 The Author(s).

Publication Source (Journal or Book title)

Human Molecular Genetics

First Page

2825

Last Page

2838

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