Authors

Georg B. Ehret, Johns Hopkins University School of Medicine
Patricia B. Munroe, Barts and The London School of Medicine and Dentistry
Kenneth M. Rice, University of Washington
Murielle Bochud, Institut Universitaire de Médecine Sociale et Préventive Lausanne
Andrew D. Johnson, Framingham Heart Study
Daniel I. Chasman, Brigham and Women's Hospital
Albert V. Smith, Icelandic Heart Association
Martin D. Tobin, University of Leicester
Germaine C. Verwoert, Erasmus MC
Shih Jen Hwang, Framingham Heart Study
Vasyl Pihur, Johns Hopkins University School of Medicine
Peter Vollenweider, Centre Hospitalier Universitaire Vaudois
Paul F. O'Reilly, Imperial College London
Najaf Amin, Erasmus MC
Jennifer L. Bragg-Gresham, University of Michigan School of Public Health
Alexander Teumer, Universität Greifswald
Nicole L. Glazer, University of Washington School of Medicine
Lenore Launer, National Institute on Aging (NIA)
Jing Hua Zhao, Wellcome-MRC Institute of Metabolic Science
Yurii Aulchenko, Erasmus MC
Simon Heath, Centre National de Recherche en Génomique Humaine
Siim Sober, Tartu Ülikool
Afshin Parsa, University of Maryland School of Medicine
Jian'an Luan, Wellcome-MRC Institute of Metabolic Science
Pankaj Arora, Massachusetts General Hospital
Abbas Dehghan, Erasmus MC
Feng Zhang, King's College London
Gavin Lucas, Parc de Recerca Biomèdica de Barcelona
Andrew A. Hicks, Eurac Research
Anne U. Jackson, University of Michigan, Ann Arbor
John F. Peden, The Wellcome Centre for Human Genetics
Toshiko Tanaka, National Institute on Aging (NIA)
Sarah H. Wild, The University of Edinburgh

Document Type

Article

Publication Date

10-6-2011

Abstract

Blood pressure is a heritable trait influenced by several biological pathways and responsive to environmental stimuli. Over one billion people worldwide have hypertension (≥140mmg Hg systolic blood pressure ≥90mmg Hg diastolic blood pressure). Even small increments in blood pressure are associated with an increased risk of cardiovascular events. This genome-wide association study of systolic and diastolic blood pressure, which used a multi-stage design in 200,000 individuals of European descent, identified sixteen novel loci: six of these loci contain genes previously known or suspected to regulate blood pressure (GUCY1A3 GUCY1B3, NPR3 C5orf23, ADM, FURIN FES, GOSR2, GNAS EDN3); the other ten provide new clues to blood pressure physiology. A genetic risk score based on 29 genome-wide significant variants was associated with hypertension, left ventricular wall thickness, stroke and coronary artery disease, but not kidney disease or kidney function. We also observed associations with blood pressure in East Asian, South Asian and African ancestry individuals. Our findings provide new insights into the genetics and biology of blood pressure, and suggest potential novel therapeutic pathways for cardiovascular disease prevention. © 2011 Macmillan Publishers Limited. All rights reserved.

Publication Source (Journal or Book title)

Nature

First Page

103

Last Page

109

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