Authors

Cristen J. Willer, University of Michigan Medical School
Ellen M. Schmidt, University of Michigan Medical School
Sebanti Sengupta, University of Michigan, Ann Arbor
Gina M. Peloso, School of Public Health
Stefan Gustafsson, Uppsala Universitet
Stavroula Kanoni, Wellcome Sanger Institute
Andrea Ganna, Uppsala Universitet
Jin Chen, University of Michigan, Ann Arbor
Martin L. Buchkovich, UNC School of Medicine
Samia Mora, Brigham and Women's Hospital
Jacques S. Beckmann, Centre Hospitalier Universitaire Vaudois
Jennifer L. Bragg-Gresham, University of Michigan, Ann Arbor
Hsing Yi Chang, National Health Research Institutes Taiwan
Ayse Demirkan, Erasmus MC
Heleen M. Den Hertog, Erasmus MC
Ron Do, Massachusetts General Hospital
Louise A. Donnelly, University of Dundee School of Medicine
Georg B. Ehret, Hôpitaux Universitaires de Genève
Tõnu Esko, Broad Institute
Mary F. Feitosa, Washington University School of Medicine in St. Louis
Teresa Ferreira, The Wellcome Centre for Human Genetics
Krista Fischer, Tartu Ülikooli Genoomika Instituut
Pierre Fontanillas, Broad Institute
Ross M. Fraser, The University of Edinburgh
Daniel F. Freitag, University of Cambridge
Deepti Gurdasani, Wellcome Sanger Institute
Kauko Heikkilä, Helsingin Yliopisto
Elina Hyppönen, UCL Great Ormond Street Institute of Child Health
Aaron Isaacs, Erasmus MC
Anne U. Jackson, University of Michigan, Ann Arbor
Åsa Johansson, Uppsala Universitet
Toby Johnson, Barts and The London School of Medicine and Dentistry
Marika Kaakinen, Oulun Yliopisto

Document Type

Article

Publication Date

11-1-2013

Abstract

Levels of low-density lipoprotein (LDL) cholesterol, high-density lipoprotein (HDL) cholesterol, triglycerides and total cholesterol are heritable, modifiable risk factors for coronary artery disease. To identify new loci and refine known loci influencing these lipids, we examined 188,577 individuals using genome-wide and custom genotyping arrays. We identify and annotate 157 loci associated with lipid levels at P < 5 × 10 -8, including 62 loci not previously associated with lipid levels in humans. Using dense genotyping in individuals of European, East Asian, South Asian and African ancestry, we narrow association signals in 12 loci. We find that loci associated with blood lipid levels are often associated with cardiovascular and metabolic traits, including coronary artery disease, type 2 diabetes, blood pressure, waist-hip ratio and body mass index. Our results demonstrate the value of using genetic data from individuals of diverse ancestry and provide insights into the biological mechanisms regulating blood lipids to guide future genetic, biological and therapeutic research. © 2013 Nature America, Inc. All rights reserved.

Publication Source (Journal or Book title)

Nature Genetics

First Page

1274

Last Page

1285

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