Authors

Georg B. Ehret, Johns Hopkins University School of Medicine
Teresa Ferreira, The Wellcome Centre for Human Genetics
Daniel I. Chasman, Brigham and Women's Hospital
Anne U. Jackson, University of Michigan, Ann Arbor
Ellen M. Schmidt, University of Michigan Medical School
Toby Johnson, Barts and The London School of Medicine and Dentistry
Gudmar Thorleifsson, deCODE genetics
Jian'An Luan, School of Clinical Medicine
Louise A. Donnelly, University of Dundee
Stavroula Kanoni, Barts and The London School of Medicine and Dentistry
Ann Kristin Petersen, Helmholtz Center Munich German Research Center for Environmental Health
Vasyl Pihur, Johns Hopkins University School of Medicine
Rona J. Strawbridge, Karolinska Institutet
Dmitry Shungin, Umeå Universitet
Maria F. Hughes, Queen's University Belfast
Osorio Meirelles, National Institute on Aging (NIA)
Marika Kaakinen, Hammersmith Hospital
Nabila Bouatia-Naji, PARCC - Paris-Centre de Recherche Cardiovasculaire
Kati Kristiansson, Terveyden ja Hyvinvoinnin Laitos
Sonia Shah, University College London
Marcus E. Kleber, Universität Heidelberg
Xiuqing Guo, Harbor-UCLA Medical Center
Leo Pekka Lyytikäinen, Fimlab Laboratoriot Oy
Cristiano Fava, Lunds Universitet
Niclas Eriksson, Uppsala Universitet
Ilja M. Nolte, Universitair Medisch Centrum Groningen
Patrik K. Magnusson, Karolinska Institutet
Elias L. Salfati, Stanford University School of Medicine
Loukianos S. Rallidis, School of Medicine
Elizabeth Theusch, Children's Hospital Oakland Research Institute
Andrew J.P. Smith, University College London
Lasse Folkersen, Karolinska Institutet
Kate Witkowska, Barts and The London School of Medicine and Dentistry

Document Type

Article

Publication Date

10-1-2016

Abstract

To dissect the genetic architecture of blood pressure and assess effects on target organ damage, we analyzed 128,272 SNPs from targeted and genome-wide arrays in 201,529 individuals of European ancestry, and genotypes from an additional 140,886 individuals were used for validation. We identified 66 blood pressure-associated loci, of which 17 were new; 15 harbored multiple distinct association signals. The 66 index SNPs were enriched for cis-regulatory elements, particularly in vascular endothelial cells, consistent with a primary role in blood pressure control through modulation of vascular tone across multiple tissues. The 66 index SNPs combined in a risk score showed comparable effects in 64,421 individuals of non-European descent. The 66-SNP blood pressure risk score was significantly associated with target organ damage in multiple tissues but with minor effects in the kidney. Our findings expand current knowledge of blood pressure-related pathways and highlight tissues beyond the classical renal system in blood pressure regulation.

Publication Source (Journal or Book title)

Nature Genetics

First Page

1171

Last Page

1184

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