Authors

Vincent Careau, University of Ottawa
Lewis G. Halsey, University of Roehampton
Herman Pontzer, Duke University
Philip N. Ainslie, Liverpool John Moores University
Lene F. Andersen, Universitetet i Oslo
Liam J. Anderson, University of Birmingham
Lenore Arab, David Geffen School of Medicine at UCLA
Issad Baddou, Université Ibn Tofail
Kweku Bedu-Addo, Kwame Nkrumah University of Science & Technology
Ellen E. Blaak, Universiteit Maastricht
Stephane Blanc, University of Georgia
Alberto G. Bonomi, Philips Research
Carlijn V.C. Bouten, Technische Universiteit Eindhoven
Maciej S. Buchowski, Vanderbilt University Medical Center
Nancy F. Butte, USDA ARS Children's Nutrition Research Center
Stefan G.J.A. Camps, Universiteit Maastricht
Graeme L. Close, Liverpool John Moores University
Jamie A. Cooper, University of Georgia
Sai Krupa Das, Jean Mayer USDA Human Nutrition Research Center on Aging
Richard Cooper, Loyola University Chicago
Lara R. Dugas, Loyola University Chicago
Simon D. Eaton, University College London
Ulf Ekelund, Norges Idrettshøgskole
Sonja Entringer, Charité – Universitätsmedizin Berlin
Terrence Forrester, The University of the West Indies
Barry W. Fudge, University of Glasgow
Annelies H. Goris, Universiteit Maastricht
Michael Gurven, University of California, Santa Barbara
Catherine Hambly, University of Aberdeen
Asmaa El Hamdouchi, Université Ibn Tofail
Marije B. Hoos, Universiteit Maastricht
Sumei Hu, Institute of Genetics and Developmental Biology Chinese Academy of Sciences
Noorjehan Joonas, Ministry of Health

Document Type

Article

Publication Date

10-25-2021

Abstract

Understanding the impacts of activity on energy balance is crucial. Increasing levels of activity may bring diminishing returns in energy expenditure because of compensatory responses in non-activity energy expenditures.1–3 This suggestion has profound implications for both the evolution of metabolism and human health. It implies that a long-term increase in activity does not directly translate into an increase in total energy expenditure (TEE) because other components of TEE may decrease in response—energy compensation. We used the largest dataset compiled on adult TEE and basal energy expenditure (BEE) (n = 1,754) of people living normal lives to find that energy compensation by a typical human averages 28% due to reduced BEE; this suggests that only 72% of the extra calories we burn from additional activity translates into extra calories burned that day. Moreover, the degree of energy compensation varied considerably between people of different body compositions. This association between compensation and adiposity could be due to among-individual differences in compensation: people who compensate more may be more likely to accumulate body fat. Alternatively, the process might occur within individuals: as we get fatter, our body might compensate more strongly for the calories burned during activity, making losing fat progressively more difficult. Determining the causality of the relationship between energy compensation and adiposity will be key to improving public health strategies regarding obesity.

Publication Source (Journal or Book title)

Current Biology

First Page

4659

Last Page

4666.e2

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